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Estrogen is implicated in the development and differentiation of several tissues, including bone. To investigate potential mechanisms for estrogen-dependent changes in bone growth, the expression of the estrogen receptor-1 (ER-1) gene was examined in developing mouse tibiae by in situ hybridization. The ER-1 mRNA signal was localized to mesenchyme within the perichondrium, as well as to preosteoblast cells at specific stages of matrix deposition. These data suggest that increased ER-1 gene expression in preosteoblast cells and mesenchyme may be functionally related to estrogen-dependent increases in bone growth. and places where they historically had to fight for the essentials. Some of these things were done to “hide” information about risk — after all, if you have to label a substance as a carcinogen, you have to admit it is carcinogenic. We still need to know more about the risks of alcohol use, but we also need to know that it is carcinogenic.
If a man drinks alcohol, he also drinks alcohol, period. If a woman wants a socially accepted beverage, she will take the time to be “perfect” in the process. “Perfect” means socially approved and “real,” too. It’s
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We have previously shown that a single large dose (60 min) of carbon monoxide (CO) may cause lung damage in rats that is associated with free-radical generation. However, the involvement of lipid peroxidation (LP) in this type of injury has not been evaluated. We therefore administered 60, 45, and 15 mg/kg of CO to rats and measured the levels of LP in lung tissue, blood, plasma, and bronchoalveolar lavage fluid (BALF) at 1, 2, 4, 8, 12, and 24 h after CO exposure. Lung tissue levels of malondialdehyde (MDA) and 4-hydroxyalkenals significantly increased immediately after the administration of 60 and 45 mg of CO. At that time-point