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Why isn’t using entities harder to test than using objects?
I was reading an article about testing, and I read that it is hard to test a non-readable method, so that’s why programmers tend to use objects (objects make it easier to test). However, I was wondering why isn’t using entities that much harder than using objects? I mean, I can see they are easier to edit and to use, so why do you make such a big deal of testing them? Is it that only when they have methods that do not have any other reasonable way of using them (for example, trying to find the problem before finding out that the code has no valid business rule), they require testing?
A:
Entities are hard to test for a very simple reason: they are too big. By “big” I mean it is virtually impossible to test them all at once. You need to break them down into smaller parts that you can test at once. This is easier when testing with objects and one object at a time. On the other hand, if entities are too big, you can’t be sure that every field is correctly validated, whether a getter or setter is actually overriden, etc.
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In vitro mutagenicity and cytotoxicity of deoxynucleoside 3-methyl-5-beta-d-riboside (3-Me-5′-rD), deoxycytidine 3-methyl-5-beta-d-riboside (3-Me-5′-dC), 2′-deoxyuridine 3-methyl-5-beta-d-riboside (3-Me-5′-U), 2′-deoxyadenosine 3-methyl-5-beta-d-riboside (3-Me-5′-Ade), 2-aminoadenine, and 3-aminoadenine were measured in the HPRT- and L5178Y TK-assays. All modifications result in a considerably enhanced mutagenicity in the HPRT-test. A significant, positive correlation was observed between mutagenicity in both assays, but cytotoxicity in the L5178Y TK-assay was not detected. The mutagenicity of 3-Me-5′-rD, 3-Me-5′-Ade, and 2-aminoadenine were increased at concentrations up to 10(-3) M. However, cytotoxicity was detected only at concentrations of 10(-4) M. 3-Me-5′-U and 3-Me-5′-dC were highly toxic at all concentrations investigated. UV-induced mutations at the hypoxanthine-guanine transition points were measured in the HPRT- and L5178Y TK-assays using Hprt- mutants of R1P2 strains. UV-induced mutation frequencies were only detectable in the HPRT-test. At all concentrations tested, UV-induced mutation frequency in the HPRT-test correlated well with mutagenicity in the HPRT-test. However, UV-induced mutation frequencies in the HPRT-test were considerably lower than mutagenicity in the L5178Y TK-test, indicating that mutagenicity in the L5178Y TK-test is due to the absence of repair processes.How to